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119 These results suggest that ACSL4 plays a critical role in regulating ferroptosis in glioma cells, indicating its potential as a therapeutic target for enhancing ferroptosis-based treatments in glioma management

No confirmed evidence from published studies demonstrates that BPC-157 causes cancer, though its angiogenic mechanism creates a theoretical risk of accelerating growth in pre-existing tumors through increased vascular supply

In human cancer epithelial cell lines, such as colorectal and prostate cancer cells, mTORC1 promotes the utilization of glutamine by activating GLUD [98]