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BPC-157 acts predominantly through angiogenic and growth-factor receptor pathways, including VEGFR2 upregulation, FAK-paxillin pathway activation, and eNOS-driven vascular effects

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In experimental systems, the combination is studied as a model for examining how complementary peptide signaling pathways influence tissue remodeling and cellular repair responses

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They are frequently investigated together because their potential mechanisms of action are thought to be complementary, creating a synergistic effect that is a popular subject of preclinical studies