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Together, these results indicate that decreased GSH/GSSG redox/antioxidant capacity and increased oxidative stress in the autism brain may have functional consequence in terms of a chronic inflammatory response, increased mitochondrial superoxide production, and oxidative protein and DNA damage

Consistent results were observed in a subanalysis of OS by high-dose steroid use in the durvalumab arm among participants who experienced an imAE, and interpretation of these results is again limited by the small number of participants within each subgroup

However, it does not stop: Hydrolysis Oxidation Deamidation Natural chemical degradation Chemical stability and microbial protection are not the same thing

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DOI: 10.1016/j.biopha.2015.12.024 26