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The generation of ferroptosis signals requires the formation of CoA derivatives of PUFAs and their binding to phospholipids, which represents a potential target for treating diseases associated with ferroptosis
doi: 10.1016/j.bcp.2021.114661 70 HinoKYanatoriIHaraYNishinaS
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Ketone bodies and ketogenic diets may exert neuroprotective effects by enhancing anti-apoptotic and antioxidant defences (e.g., via inhibition of mPTP opening, mitochondrial ROS production, and caspase activation, and activation of SIRT1/2/3PGC1FOXO1/3a and ERK/CREB/eNOS pathways, with Nrf2 engagement), and by modulating NAD-dependent signalling and epigenetic/post-translational regulation (including HDAC/p300-mediated histone modifications) that can upregulate neurotrophic programs such as BDNF expression [69]
NDUFV2 and NDUFS1 are nuclear-encoded complex I subunits, involving in mitochondrial oxidative phosphorylation 21
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