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Furthermore, butyrate itself upregulates the expression of MCT1 and MCT4 [78, 84], creating a positive feedback loop that enhances the cellular uptake of their substrates, including butyrate

Sandstrom et al., 2017), solitary nucleus, and catecholaminergic neurons in the pituitary-adrenal system (Edmonds and Edwards, Studies have indicated that PQ dopaminergic neurotoxicity is associated with increased cellular oxidative stress and neuroinflammation, which induces -synuclein aggregation and impairs neurons (Manning-Bog et al., 2002

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Off-target toxicity limits the maximum tolerated dose of chemotherapeutic drugs and causes long term health problems in cancer survivors, including an acceleration of aging (Henderson et al., 2014)