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Benzbromarone and probenecid inhibit URAT1-mediated reabsorption in the proximal convoluted tubules, thereby increasing uric acid excretion (144)

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The hepatotoxicity associated with acetaminophen is primarily due to the formation of a reactive metabolite, N-acetyl-p-benzoquinoneimine (NAPQI), which induces oxidative stress and cellular damage in susceptible species, including dogs, cats, non-human primates, ferrets, birds, and pigs (Ben-Shachar et al., 2012)

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Impaired activity of the ubiquitinproteasome system in transgenic mice expressing ALS/dementia-linked mutant UBQLN2 (P02.170) Neurology